SLC39A8-mediated zinc dyshomeostasis potentiates kidney disease
https://www.profitableratecpm.com/f4ffsdxe?key=39b1ebce72f3758345b2155c98e6709c
Proceedings of the National Academy of Sciences, Volume 122, number 38, September 2025.
ImmonanceThrough Integrative Analysis of UK Biobank Data combined with multimodal experimental studies in genetically modified Knockout / Transgenic mice and renal lesions models, we have identified the SLC39A8 A391T variant as a protest genetic modifier …
